Inner ear injuries are the cause of labyrinthine traumatic syndrome, which is a set of specific signs of dysfunction of the sound and vestibular analyzers, combined with possible general and focal lesions of the brain.
Hysteria is a special form of neurosis, manifested by various functional mental, somatic and neurological disorders, developing in individuals with a special structure of the nervous system, but also occurring in healthy people under certain conditions (weakening of the nervous system under the influence of psychogenic and somatogenic pathological factors).
Central vestibular syndromes occur when neurons and pathways of the vestibular analyzer are damaged, starting from the vestibular nuclei and ending with the cortical zones of this analyzer, as well as when similar damage occurs to brain structures adjacent to the central vestibular structures.
Senile hearing loss, or presbycusis, along with presbyopia, is the most common manifestation of involutional processes in the aging organism, manifested in the withering of all its functions and, above all, metabolic processes in the nervous system.
As a clinical phenomenon, this syndrome has been described by many authors. The absence of a clear etiological cause for this sudden one- or two-sided deafness has caused much discussion among audiologists, which, however, has not led to any results.
Acute acoustic trauma occurs as a result of the impact on the hearing organ of powerful impulse noise of more than 160 dB, often in combination with a sharp increase in barometric pressure during an explosion.
Acoustic trauma occurs as a result of prolonged or impulse noise on the organ of hearing or vibration, exceeding the permissible norms in intensity or the tolerance of the receptor structures of the inner ear to these stimuli.
Congenital degenerative cochleopathy (congenital deafness) is caused by prenatal or intranatal pathogenic factors, manifested by deafness from the moment of birth.
Neomycin selectively acts on the hair cells of the cochlea and often causes more frequent and profound hearing loss than streptomycin, up to and including complete deafness.
The pathogenesis of streptomycin toxic-degenerative labyrinthosis is based on the antibacterial property of this drug, which consists of its penetration into the microbial, as well as the receptor cell, and binding to specific receptor proteins of their ribosomes.
